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Theoretically, the senescence process entails the complications of fueling inflammation and disrupting the tissues structure and function

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In non-infectious, sterile-inflammatory contexts such as metabolic disease, thrombosis, or PCOS, NETosis can be initiated through oxidized lipid species and platelet-derived cues rather than microbial PAMPs (54, 57)

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Consequences of hyperoxia and the toxicity of oxygen in the lung